Advances in Cancer Research, Vol. 56 by GEORGE F. & GEORGE KLIEN (eds.) VANDE WOUDE

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By GEORGE F. & GEORGE KLIEN (eds.) VANDE WOUDE

Meant for simple and medical researchers, immunologists, virologists and molecular biologists, this publication is a part of a chain which provides serious overviews on chosen elements of either melanoma study and the elemental underlying sciences.

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B. (1989). Oncogene 4,549-558. , and. Leder, P. (1983). )34,779-787. , Nordan, R. , Owens, J. , Marcu, K. , and Mushinski, J. F. (1989). Oncogene 4,615-623. Beer, D. , Zweifel, K. , Simpson, D. , and Pitot, H. C. J. Cell. Physiol. 131, 29-35. Bentley, D. , and Groudine, M. Nafure (London) 321,702-706. Bentley, D. , and Groudine, M. Mol. Cell. B i d . 6, 3481-3489. Bentley, D. , Brown, W. , and Groudine, M. (1989). Genes Dev. 3, 1179- I 189. , and Adams, J. M. (1983). EMBOJ. 2, 2375-2383. , Jansen, H.

Loss of the block to transcription elongation in c-myc exon 1 5 . In the translocated alleles that maintain the first exon, a shift in promoter utilization to increased use of the minor upstream promoter, P I 6. Loss of DNase I hypersensitive sites in the nontranslocated c-myc allele, but maintainance of these same sites in the translocated allele 1. Abnormal Gene Topography in Burkitt’s Lymphoma A striking feature of BL cells is the consistent presence of reciprocal translocations between the c-myc locus on chromosome 8 and one of the immunoglobulin loci on chromosomes 14,2, or 22 (reviewed in Varmus, 1984; Rabbitts, 1985; Cole, 1986; Cory, 1986).

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